PROPOXUR
CASRN: 114-26-1
For other data, click on the Table of Contents

Human Health Effects:

Human Toxicity Excerpts:

ADULT VOLUNTEER ... INGESTED 1.5 MG/KG ... EXPERIENCED PROMPT FALL IN RED CELL CHOLINESTERASE ACTIVITY THAT REACHED MIN OF 27% OF NORMAL IN 15 MIN. THIS WAS QUICKLY FOLLOWED BY BLURRED VISION, NAUSEA, PALLOR, SWEATING, TACHYCARDIA & VOMITING. ALL SYMPTOMS ... ABATED & ENZYME LEVELS ... NORMAL BY 2 HR ... .
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial Products. 5th ed. Baltimore: Williams and Wilkins, 1984.,p. III-87]**PEER REVIEWED**

Symptomatology: 1. Nausea, vomiting, abdominal cramps, diarrhea & excessive salivation, ... sweating. 2. Lassitude & weakness. 3. Rhinorrhea and sensation of tightness in chest may occur with inhalation exposure. 4. Blurring or dimness of vision. Miosis, ... tearing, ciliary muscle spasm, loss of accommodation and ocular pain. None of these ... signs ... is dependable for diagnosis. Mydriasis may be seen ... 5. Loss of muscle coordinatin, slurring of speech, fasciculation & twitching of muscles. 6. Difficulty in breathing, excessive secretions of saliva and of resp tract mucus, oronasal frothing, cyanosis, pulmonary rales & rhonchi, and hypertension. 7. ... Jerky movements, incontinence, convulsions and coma. 8. Death ... due to resp arrest of central origin, paralysis of resp muscles, intense bronchoconstriction or all three. /Carbaryl/
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial Products. 5th ed. Baltimore: Williams and Wilkins, 1984.,p. II-309]**PEER REVIEWED**

HUMAN ADULTS HAVE INGESTED SINGLE DOSES OF 90 MG APPARENTLY WITHOUT SYMPTOMS.
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial Products. 5th ed. Baltimore: Williams and Wilkins, 1984.,p. II-309]**PEER REVIEWED**

DURING WIDE USAGE IN MALARIA CONTROL ACTIVITIES BY WORLD HEALTH ORGANIZATION, ONLY A FEW MILD CASES OF POISONING ... NOTED.
[American Conference of Governmental Industrial Hygienists. Documentation of the Threshold Limit Values and Biological Exposure Indices. 5th ed. Cincinnati, OH:American Conference of Governmental Industrial Hygienists, 1986. 499]**PEER REVIEWED**

Moderate but transient toxicity has also been observed following exposure to a few of the more potent carbamate ester insecticides such as ... propoxur (Baygon) ...
[Amdur, M.O., J. Doull, C.D. Klaasen (eds). Casarett and Doull's Toxicology. 4th ed. New York, NY: Pergamon Press, 1991. 584]**PEER REVIEWED**

Medical Surveillance:

The clinical picture is similar to the muscarinic and nicotinic effects of organophosphates, but usually is less severe. Symptoms develop within 15 minutes to 2 hours and last several hours unless continued absorption occurs from clothing. Blurred vision, nausea, vomiting, abdominal cramps, salivation, and diaphoresis are common. Dyspnea, tremors, muscle twitching, ataxia, and headache also appear. Symptom beyond 24 hours probably do not result from carbamate intoxication. /Carbamate/
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1077]**PEER REVIEWED**

 

Probable Routes of Human Exposure:

People may be exposed to propoxur in indoor air via inhalation or dermal contact where it is used to control cockroaches, flies and mosquitoes and outdoors when it is used to control lawn and turf insects(1). Exposure would be particularly high indoors(SRC).
[(1) Lewis RG et al; Environ Monit Assess 10: 59-73 (1988)]**PEER REVIEWED**

Propoxur was detected in 6 of 9 personal air samples of retired or semiretired persons in a pilot project of pesticide exposure(3). The concentrations measured ranged up to 0.60 ug/cu m, 0.10 ug/cu m, mean. Outside these houses, on the patio or porch, 4 of the nine samples contained propoxur ranging up to 0.0039 ug/cu m, 0.0034 ug/cu m, mean.
[(1) Lewis RG et al; Environ Monit Assess 10: 59-73 (1988)]**PEER REVIEWED**

 

Emergency Medical Treatment:

 

 

Emergency Medical Treatment:

 

EMT Copyright Disclaimer:
Portions of the POISINDEX(R) database are provided here for general reference. THE COMPLETE POISINDEX(R) DATABASE, AVAILABLE FROM MICROMEDEX, SHOULD BE CONSULTED FOR ASSISTANCE IN THE DIAGNOSIS OR TREATMENT OF SPECIFIC CASES. Copyright 1974-1998 Micromedex, Inc. Denver, Colorado. All Rights Reserved. Any duplication, replication or redistribution of all or part of the POISINDEX(R) database is a violation of Micromedex' copyrights and is strictly prohibited.

The following Overview, *** CARBAMATE INSECTICIDES ***, is relevant for this HSDB record chemical.

Life Support:
  o   This overview assumes that basic life support measures
      have been instituted.                           
Clinical Effects:
  SUMMARY OF EXPOSURE
   0.2.1.1 ACUTE EXPOSURE
     o   Carbamates are well absorbed from all routes of
         exposure.  Exposure may lead to cholinergic crisis with
         increased salivation, lacrimation, urinary
         incontinence, diarrhea, gastrointestinal cramping, and
         emesis (SLUDGE syndrome).  The syndrome may be
         indistinguishable from that seen after organophosphate
         poisoning.
     o   The usual cause of death is respiratory failure.
         Predominant serious toxicity is related to central
         nervous system depression and nicotinic effects.  CNS
         effects may include stupor, coma, seizures and
         hypotonicity.  Hypertension and tachycardia or
         cardiorespiratory depression may occur.
     o   Children may be more likely to develop CNS depression,
         seizures and hypotonia than typical SLUDGE syndrome.
         Absence of classic muscarinic  effects has been
         reported in a series of 36 children intoxicated with
         carbamate insecticides.
  HEENT
   0.2.4.1 ACUTE EXPOSURE
     o   Miosis and blurred vision are common.  Mydriasis may
         occur.
  CARDIOVASCULAR
   0.2.5.1 ACUTE EXPOSURE
     o   Bradycardias, ST depression, tachycardias, and
         hypertension have been reported.
  RESPIRATORY
   0.2.6.1 ACUTE EXPOSURE
     o   Dyspnea, wheezing, rales, increased bronchial
         secretions, respiratory muscle weakness and respiratory
         failure may occur.  Usual cause of death  is
         respiratory failure.
     o   Aspiration pneumonitis may occur.
     o   Laryngeal irritation and associated cough is common
         following inhalation of dusting powders.
  NEUROLOGIC
   0.2.7.1 ACUTE EXPOSURE
     o   In severe poisoning, respiratory depression, mental
         confusion, unconsciousness, brain hemorrhages, and
         seizures may occur.  Children may be more susceptible
         to seizures than adults.
     o   Headache, blurred vision, tremor, paresis, mental
         depression, coma, delayed neuropathies, various
         dystonias, weakness, muscle twitching may be noted.
  GASTROINTESTINAL
   0.2.8.1 ACUTE EXPOSURE
     o   Nausea, vomiting, diarrhea, and abdominal cramping have
         been reported and are common.
     o   Pancreatitis has been reported.
  HEMATOLOGIC
   0.2.13.1 ACUTE EXPOSURE
     o   Disseminated intravascular coagulation has been
         reported in humans.  Brain hemorrhages were seen in one
         case.  Animals have experienced decreased hemoglobin,
         RBC count, platelets, and erythrocyte sedimentation.
  DERMATOLOGIC
   0.2.14.1 ACUTE EXPOSURE
     o   A possible occupational effect is contact dermatitis.
         Diaphoresis may be seen after exposure.  Cellulitis was
         seen after injection of household spray.
  MUSCULOSKELETAL
   0.2.15.1 ACUTE EXPOSURE
     o   Rhabdomyolysis may occur.                       
Laboratory:
  o   Determine plasma and red blood cell cholinesterase.
  o   Obtain a chest x-ray in symptomatic patients.      
Treatment Overview:
  ORAL EXPOSURE
    o   Emesis is NOT recommended because of the possibility of
        seizures or respiratory depression developing prior to
        or during emesis.
    o   ACTIVATED CHARCOAL:  Administer charcoal as slurry (240
        mL water/30 g charcoal).  Usual dose:  25 to 100 g in
        adults/adolescents, 25 to 50 g in children (1 to 12
        years), and 1 g/kg in infants less than 1 year old.
    o   GASTRIC LAVAGE:  Consider after ingestion of a
        potentially life-threatening amount of poison if it can
        be performed soon after ingestion (generally within 1
        hour).  Protect airway by placement in Trendelenburg and
        left lateral decubitus position or by endotracheal
        intubation.  Control any seizures first.
     1.  CONTRAINDICATIONS:  Loss of airway protective reflexes
         or decreased level of consciousness in unintubated
         patients; following ingestion of corrosives;
         hydrocarbons (high aspiration potential); patients at
         risk of hemorrhage or gastrointestinal perforation; and
         trivial or non-toxic ingestion.
    o   ADMINISTER ATROPINE SULFATE - in repeated doses
        intravenously until atropinization is achieved
        (indicated by drying of pulmonary secretions).
     1.  ADULT DOSE - 2 to 4 milligrams every 10 to 15 minutes.
     2.  CHILD - 0.05 milligram/kilogram every 10 to 15 minutes.
    o   Administer pralidoxime if severe toxicity develops.
     1.  PRALIDOXIME (Protopam, 2-PAM):  Treat moderate to
         severe poisoning (fasciculations, muscle weakness,
         respiratory depression, coma, seizures) with 2-PAM in
         addition to atropine; most effective if given within 48
         hours, but has had efficacy up to 6 days.  May require
         administration for several days.
      a.  INITIAL DOSE:  ADULT:  1 to 2 g in 100 to 150 ml 0.9%
          saline IV over 30 min.   CHILD:  20 to 50 mg/kg as a
          5% solution IV over 30 min.
      b.  Repeat these doses in 1 hour and then every 6 to 12
          hours if  muscle weakness or fasciculations persist,
          or begin continuous infusion.
      c.  CONTINUOUS INFUSION:  Administer as a 2.5% solution in
          0.9% saline.  ADULT:   500 mg/hour.  CHILD:  9 to 19
          mg/kg/hour.
    o   SEIZURES:  Administer a benzodiazepine IV; DIAZEPAM
        (ADULT:  5 to 10 mg,  repeat every 10 to 15 min as
        needed.  CHILD:  0.2 to 0.5 mg/kg, repeat every  5 min
        as needed) or LORAZEPAM (ADULT:  4 to 8 mg; CHILD:  0.05
        to 0.1 mg/kg).
     1.  Consider phenobarbital if seizures recur after diazepam
         30 mg (adults)  or 10 mg (children > 5 years).
     2.  Monitor for hypotension, dysrhythmias, respiratory
         depression, and need  for endotracheal intubation.
         Evaluate for hypoglycemia, electrolyte disturbances,
         hypoxia.
  INHALATION EXPOSURE
    o   INHALATION:  Move patient to fresh air.  Monitor for
        respiratory distress.  If cough or difficulty breathing
        develops, evaluate for respiratory tract irritation,
        bronchitis, or pneumonitis.  Administer oxygen and
        assist ventilation as required.  Treat bronchospasm with
        beta2  agonist and corticosteroid aerosols.
  EYE EXPOSURE
    o   DECONTAMINATION:  Irrigate exposed eyes with copious
        amounts of tepid water for at least 15 minutes.  If
        irritation, pain, swelling, lacrimation, or photophobia
        persist, the patient should be seen in a health care
        facility.
  DERMAL EXPOSURE
    o   DECONTAMINATION:  Remove contaminated clothing and
        jewelry.  Wash  the skin, including hair and nails,
        vigorously; do repeated soap washings.  Discard
        contaminated clothing.
Range of Toxicity:
  o   Acute toxicity depends on kinetics of absorption ie,
      sudden absorption of a low toxicity compound may have a
      greater effect.  Aldicarb is considered the most toxic
      carbamate.                                             


[Rumack BH: POISINDEX(R) Information System. Micromedex, Inc., Englewood, CO, 2001; CCIS Volume 110, edition exp November, 2001. Hall AH & Rumack BH (Eds):TOMES(R) Information System. Micromedex, Inc., Englewood, CO, 2001; CCIS Volume 110, edition exp November, 2001.] **PEER REVIEWED**

 

Antidote and Emergency Treatment:

The clinical approach to carbamate toxicity is similar to that for organophosphate poisoning; the major exception is that pralidoxime usually is not recommended. /Carbamates/
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1077]**PEER REVIEWED**

Stabilization: Assess the adequacy of the airway and ventilation and use oxygen, suction, intubation, artificial ventilation, intravenous lines, and cardiac monitors as needed. /Carbamates/
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1078]**PEER REVIEWED**

Decontamination: The usual measures of decontamination (ipecac/lavage, charcoal, cathartics) may be used when the patient presents within 2-4 hours of exposure. Container identification is important to determine appropriate therapeutic measures, since the vehicle (eg, hydrocarbon, methanol) may be as toxic as the insecticide. When syrup of ipecac is used, the patient must be observed closely to prevent aspiration. In experimental animals, carbaryl was not well absorbed by activated charcoal, but use of activated charcoal is still recommended because of its freedom from side effects. Be sure to remove contaminated clothing and wash.
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1078]**PEER REVIEWED**

Elimination Enhancement: No methods are recommended, because of the short clinical effect of carbamates and the presence of an effective antidote (atropine). /Carbamates/
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1078]**PEER REVIEWED**

Antidotes: Atropine is the antidote of choice as in organophosphate poisoning. Although the total amount of atropine required usually is less, the same initial doses are recommended. Pralidoxime usually is unnecessary and may reduce the effectiveness of atropine (especially with carbaryl). ... Patients require approximately 6-12 hours of atropine treatment, but all significantly poisoned patients should be observed at least 24 hours after the last atropine dose. /Carbamates/
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1078]**PEER REVIEWED**

 

Animal Toxicity Studies:

 

 

Non-Human Toxicity Excerpts:

BAYGON INHIBITS CHOLINESTERASE ... HOWEVER ... INHIBITION IS RAPIDLY REVERSIBLE. FOLLOWING ACUTE, LOW-LEVEL EXPOSURES, SYMPTOMS WHICH DO OCCUR ARE TRANSITORY IN NATURE.
[American Conference of Governmental Industrial Hygienists. Documentation of the Threshold Limit Values and Biological Exposure Indices. 5th ed. Cincinnati, OH:American Conference of Governmental Industrial Hygienists, 1986. 449]**PEER REVIEWED**

... PERMANENT BEHAVIORAL ABERRATIONS /WERE DOCUMENTED/ IN RATS WHICH RECEIVED CARBARYL OR PROPOXUR REPEATEDLY ... . /CARBARYL/
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial Products. 5th ed. Baltimore: Williams and Wilkins, 1984.,p. III-88]**PEER REVIEWED**

DERMAL TOXICITY TO RATS 1000 MG/KG AS OIL SUSPENSION WITHOUT SYMPTOMS OF INTOXICATION OR IRRITATION AFTER 4 HR EXPOSURE TO SHAVED ABDOMEN. SUBCHRONIC FEEDING STUDIES ON RATS WITH 7.5 MG/KG FOR 28 CONSECUTIVE DAYS SHOWED NO TOXIC SYMPTOMS. NO INHALATION TOXICITY (1 MG/L AIR, 3 TIMES @ HOURLY INTERVALS, REPEATED 5 DAYS).
[Spencer, E. Y. Guide to the Chemicals Used in Crop Protection. 7th ed. Publication 1093. Research Institute, Agriculture Canada, Ottawa, Canada: Information Canada, 1982. 488]**PEER REVIEWED**

IN 2-YR FEEDING TRIALS MALE & FEMALE RATS RECEIVING 250 MG ACTIVE INGREDIENT/KG DIET SHOWED NO ILL EFFECT; AT 750 MG/KG DIET THE LIVER WEIGHT OF FEMALE RATS INCREASED, OTHERWISE THERE WAS NO ILL-EFFECT. IT IS HIGHTLY TOXIC TO HONEY BEES.
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 717]**PEER REVIEWED**

SIGNS OF INTOXICATION /AFTER ACUTE ORAL ADMIN TO BIRDS/: NUTATION, LACRIMATION, ATAXIA, MIOSIS, LETHARGY, ASYNERGY, IMBALANCE, SALIVATION, HYPOACTIVITY, GOOSE-STEPPING ATAXIA, FALLING, TACHYPNEA, DYSPNEA, FASCICULATION, PTOSIS, DIARRHEA, IMMOBILITY, TREMORS, CONVULSIONS, WINGS SPREAD IN TETANY, AND OPISTHOTONOS. DEPENDING ON THE SPECIES, SIGNS APPEARED AS SOON AS 5 MIN, MORTALITIES OCCURRED BETWEEN 5 AND 45 MIN OR OVERNIGHT, AND REMISSION OCCURRED FROM 90 MIN TO SEVERAL DAYS AFTER TREATMENT.
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

Propoxur was not mutagenic to six strains of Salmonella typhimurium.
[Hayes, Wayland J., Jr. Pesticides Studied in Man. Baltimore/London: Williams and Wilkins, 1982. 449]**PEER REVIEWED**

Baygon was administered IG once daily to CD rats (5 to 50 mg/kg), on the 7th-19th day of gestation or to CD-1 mice (5 to 60 mg/kg) on days 6-16 of gestation. ... Baygon was not teratogenic in the CD rat or CD-1 mouse at maternally nontoxic dose levels. ...
[Courtney KD et al; J Environ Sci Health (B) 20 (4): 373-406 (1985)]**PEER REVIEWED**

The toxicity of /propoxur/ was studied in rats. Changes in electroencephalogram and in learning ability were observed at lower concn than changes in cholinesterase inhibition and in body or organ weight. ...
[Desi I; Z Gesamte Hyg Ihre Grenzgeb 30 (6): 326-9 (1984)]**PEER REVIEWED**

Propoxur was effective only in the reduction of cholinesterase and increase of liver weight /in rats/. The latter effect was not accompanied by microsomal induction. Therefore, enzymes were not induced by propoxur, even at grossly toxic levels. Thus, propoxur can safely be recommended for use in lab animal rooms.
[Nelson DL et al; Vet Hum Toxicol 26 (4): 305-8 (1984)]**PEER REVIEWED**

The toxicity of ... propoxur decreased in rats given Aroclor 1242.
[Neskovic NK et al; Arh Hig Rada Toksikol 35 (4): 333-42 (1984)]**PEER REVIEWED**

/Propoxur/ was tested against Eisenia foetida for the purpose of using this organism as the marker species to indicate the relative toxicities of chemicals to earthworms and other soil invertebrates. The worms were exposed to deposits of /propoxur/ on filter paper for 48 hr and the mortality was recorded; concn were expressed in ug/sq cm. Based on the resulting median lethal concn values, /propoxur was/ classified as supertoxic (<1.0 ug/sq cm), extremely toxic (1-10 ug/sq cm), very toxic (10-100 ug/sq cm), moderately toxic (100-1000 ug/sq cm) or relatively nontoxic (>1000 ug/sq cm).
[Roberts BL, Dorough HW; Environ Toxicol Chem 3 (1): 67-78 (1984)]**PEER REVIEWED**

Residual toxicity to adult honeybees (Apis mellifera) of /propoxur/ sprayed on clover (Trifolium alexandrinum) at field rates recommended in Iraq was assessed by a simple labortory method in which caged bees were fed a blend of 50 clover flowers in 50 ml 25% sucrose syrup. For 6 of the insecticides tested, mortality was over 90% in bees fed with syrup blended with flowers collected on the day of spraying. Thereafter mortality declined with time to a negligible level in bees fed syrup blended with flowers collected 7 days after spraying. /Propoxur was/ compared for hazard to honeybees on the basis of the time required for mortality recorded in a 24-period to decline to 20%. With this criterion ... /propoxur/ was one of the most hazardous of the insecticides tested.
[Mansour SA, et al; J Apic Res 23 (4): 213-216 (1984)]**PEER REVIEWED**

Propoxur, a carbamate anticholinesterase pesticide, used in flea collars for dogs, caused miosis during the first week the collars were worn.
[Grant, W.M. Toxicology of the Eye. 3rd ed. Springfield, IL: Charles C. Thomas Publisher, 1986. 767]**PEER REVIEWED**

Effect in vitro of propoxur on the specific activity of calcium stimulated ATPase and calcium uptake was studied in the rat brain synaptosomes. The data suggest that propoxur might disrupt the synaptic function by altering the calcium dependent ATP hydrolysis and calcium uptake in the central nervous system.
[Babu G RV et al; Biochem Int 21 (6): 1089-94 (1990)]**PEER REVIEWED**

Modulations in ionic composition were seen in the rat brain during propoxur treatment indicating an impairment in the electric activity of neurons, oxygen consumption, ATPase system, disruption in the movement of ions across inonic pumps and synaptic transmission. The specific activity levels of ATPase were also altered confirming that the impairment in the ATPase system might be due to the ionic imbalances under propoxur stress.
[Babu G RV et al; Biochem Int 21 (6): 1105-12 (1990)]**PEER REVIEWED**

 

Non-Human Toxicity Values:

LD50 Domestic goat male 12 mo of age oral > 800 mg/kg male 12 mo of age
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Rat acute oral 95 to 104 mg/kg
[Verschueren, K. Handbook of Environmental Data of Organic Chemicals. 2nd ed. New York, NY: Van Nostrand Reinhold Co., 1983. 230]**PEER REVIEWED**

LD50 Rat dermal >1000 mg/kg
[Verschueren, K. Handbook of Environmental Data of Organic Chemicals. 2nd ed. New York, NY: Van Nostrand Reinhold Co., 1983. 230]**PEER REVIEWED**

 

Ecotoxicity Values:

LD50 Bullfrog male oral 595 mg/kg (95% confidence limit 500-707 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Canada goose male, female oral 5.95 mg/kg (95% confidence limit 4.89-7.24 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Mallard female 4-6 mo of age oral 11.9 mg/kg (95% confidence limit 10.0-14.1 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Mallard female 4-6 mo of age oral oral 9.44 mg/kg (95% confidence limit 7.49-11.9 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Sharp-tailed grouse 12-48 mo of age oral 120 mg/kg (95% confidence limit 84.8-170 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 California quail female 3-7 mo of age oral 25.9 mg/kg (95% confidence limit 14.9-45.0 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Japanese quail female 20 mo of age oral 28.3 mg/kg (95% confidence limit 20.0-40.0 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Pheasant males 3-5 mo of age oral 20.0 mg/kg (95% confidence limit 10.0-40.0 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 Chukar male, female 4-6 mo of age oral 23.8 mg/kg (95% confidence limit 20.0-28.3 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Sandhill crane male, female oral more than 60.0 mg/kg
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Rock dove male, female oral 60.4 mg/kg (95% confidence limit 38.0-96.1 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Morning dove male, female oral 4.20 mg/kg (95% confidence limit 3.54-5.00 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 14]**PEER REVIEWED**

LD50 House sparrow female adult oral 12.8 mg/kg (95% confidence limit 9.26-17.8 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 House finch male, female adult oral 3.55 mg/kg (95% confidence limit 2.25-5.69 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Dark-eyed junco male adult oral 4.76 mg/kg (95% confidence limit 4.00-5.70 mg/kg)
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LD50 Mule deer oral 100-350 mg/kg female 11 mo of age
[U.S. Department of the Interior, Fish and Wildlife Service. Handbook of Toxicity of Pesticides to Wildlife. Resource Publication 153. Washington, DC: U.S. Government Printing Office, 1984. 15]**PEER REVIEWED**

LC50 Pimephales promelas (fathead minnow) 8.8 mg/l/96 hr (confidence limit 7.4 - 10.5 mg/l), flow-through bioassay with measured concentrations, 22.5 deg C, dissolved oxygen 7.5 mg/l, hardness 45.4 mg/l calcium carbonate, alkalinity 40.0 mg/l calcium carbonate and pH 7.6.
[Geiger D.L., Call D.J., Brooke L.T. (eds). Acute Toxicities of Organic Chemicals to Fathead Minnows (Pimephales Promelas). Vol. IV. Superior Wisconsin:University of Wisconsin-Superior, 1988. 245]**PEER REVIEWED**

EC50 Pimephales promelas (fathead minnow) 9.0 mg/l/96 hr (confidence limit 7.7 - 10.5 mg/l), flow-through bioassay with measured concentrations, 22.5 deg C, dissolved oxygen 7.5 mg/l, hardness 45.4 mg/l calcium carbonate, alkalinity 40.0 mg/l calcium carbonate and pH 7.6. Effect: loss of equilibrium.
[Geiger D.L., Call D.J., Brooke L.T. (eds). Acute Toxicities of Organic Chemicals to Fathead Minnows (Pimephales Promelas). Vol. IV. Superior Wisconsin:University of Wisconsin-Superior, 1988. 245]**PEER REVIEWED**

LC50 Coturnix >5,000 ppm (95% confidence interval) /Conditions of bioassay not specified/
[Hill, E.F. and Camardese, M.B. Lethal Dietary Toxicities of Environmental Contaminants and Pesticides to Coturnix. Fish and Wildlife Technical Report 2.Washington, DC: United States Department of Interior Fish and Wildlife Service, 1986. 120]**PEER REVIEWED**

LC50 Gammarus fasciatus 50 ug/l/96 hr /Conditions of bioassay not specified/
[Verschueren, K. Handbook of Environmental Data of Organic Chemicals. 2nd ed. New York, NY: Van Nostrand Reinhold Co., 1983. 230]**PEER REVIEWED**

LC50 Chironomus riparius 64.4 ppb/24 hr /Conditions of bioassay not specified/
[Verschueren, K. Handbook of Environmental Data of Organic Chemicals. 2nd ed. New York, NY: Van Nostrand Reinhold Co., 1983. 230]**PEER REVIEWED**

LC50 Gammarus lacustris 34 ug/l/96 hr at 21 deg C (95% confidence interval), Static bioassay without aeration, pH 7.2-7.5, water hardness 40-50 mg/l as calcium carbonate and alkalinity of 30-35 mg/l.
[U.S. Department of Interior, Fish and Wildlife Service. Handbook of Acute Toxicity of Chemicals to Fish and Aquatic Invertebrates. Resource Publication No. 137. Washington, DC: U.S. Government PrintingOffice, 1980. 69]**PEER REVIEWED**

LC50 Pteronarcys 18 ug/l/96 hr at 15 deg C (95% confidence interval), Static bioassay without aeration, pH 7.2-7.5, water hardness 40-50 mg/l as calcium carbonate and alkalinity of 30-35 mg/l.
[U.S. Department of Interior, Fish and Wildlife Service. Handbook of Acute Toxicity of Chemicals to Fish and Aquatic Invertebrates. Resource Publication No. 137. Washington, DC: U.S. Government PrintingOffice, 1980. 69]**PEER REVIEWED**

LC50 Rainbow trout 8200 ug/l/96 hr at 13 deg C (95% confidence interval), Static bioassay without aeration, pH 7.2-7.5, water hardness 40-50 mg/l as calcium carbonate and alkalinity of 30-35 mg/l.
[U.S. Department of Interior, Fish and Wildlife Service. Handbook of Acute Toxicity of Chemicals to Fish and Aquatic Invertebrates. Resource Publication No. 137. Washington, DC: U.S. Government PrintingOffice, 1980. 69]**PEER REVIEWED**

LC50 Fathead minnow 25,000 ug/l/96 hr at 18 deg C (95% confidence interval), Static bioassay without aeration, pH 7.2-7.5, water hardness 40-50 mg/l as calcium carbonate and alkalinity of 30-35 mg/l.
[U.S. Department of Interior, Fish and Wildlife Service. Handbook of Acute Toxicity of Chemicals to Fish and Aquatic Invertebrates. Resource Publication No. 137. Washington, DC: U.S. Government PrintingOffice, 1980. 69]**PEER REVIEWED**

LC50 Bluegill 4800 ug/l/96 hr at 24 deg C (95% confidence interval), Static bioassay without aeration, pH 7.2-7.5, water hardness 40-50 mg/l as calcium carbonate and alkalinity of 30-35 mg/l.
[U.S. Department of Interior, Fish and Wildlife Service. Handbook of Acute Toxicity of Chemicals to Fish and Aquatic Invertebrates. Resource Publication No. 137. Washington, DC: U.S. Government PrintingOffice, 1980. 69]**PEER REVIEWED**

 

Metabolism/Pharmacokinetics:

 

 

Metabolism/Metabolites:

O-ISOPROPOXYPHENYL N-METHYLCARBAMATE YIELDS 4-HYDROXY-2-ISOPROPOXYPHENYL N-METHYLCARBAMATE, 2-HYDROXYPHENYL N-METHYLCARBAMATE, & O-ISOPROPOXYPHENYL N-HYDROXYMETHYLCARBAMATE IN BEAN. /FROM TABLE/
[Goodwin, B.L. Handbook of Intermediary Metabolism of Aromatic Compounds. New York: Wiley, 1976.,p. I-16]**PEER REVIEWED**

O-ISOPROPOXYPHENYL N-METHYLCARBAMATE YIELDS 5-HYDROXY-2-ISOPROPOXYPHENYL N-METHYLCARBAMATE, 2-HYDROXYPHENYL N-METHYLCARBAMATE, O-ISOPROPOXYPHENOL, O-ISOPROPOXYPHENYL N-HYDROXYMETHYLCARBAMATE IN RAT. /FROM TABLE/
[Goodwin, B.L. Handbook of Intermediary Metabolism of Aromatic Compounds. New York: Wiley, 1976.,p. I-16]**PEER REVIEWED**

O-ISOPROPOXYPHENYL N-METHYLCARBAMATE YIELDS O-ISOPROPOXYPHENYL N-HYDROXYMETHYLCARBAMATE IN MOUSE. /FROM TABLE/
[Goodwin, B.L. Handbook of Intermediary Metabolism of Aromatic Compounds. New York: Wiley, 1976.,p. I-16]**PEER REVIEWED**

IN ... STUDIES WITH ... MUSCA DOMESTICA L ... /METABOLITES WERE/ (IN ORDER OF DECR AMT): 5-HYDROXY-2-ISOPROPOXYPHENYL METHYLCARBAMATE, 2-HYDROXYPHENYL METHYLCARBAMATE & ACETONE, 2-ISOPROPOXYPHENYL N-HYDROXYMETHYLCARBAMATE, & 2-ISOPROPOXYPHENYL CARBAMATE. THERE WERE 6 OR MORE ADDNL UNIDENTIFIED CMPD.
[Menzie, C.M. Metabolism of Pesticides. U.S. Department of the Interior, Bureau of Sport Fisheries and Wildlife, Publication 127. Washington, DC: U.S. Government Printing Office, 1969. 50]**PEER REVIEWED**

When the tert-carbon of 2-isopropoxy group of propoxur is oxidized to form a hemiketal, mono-N-methylcarbamoylcatechol is produced as a hydrolyzed metabolite that can normally be detected as 2-isopropoxyphenol by hydrolysis of the carbamoyl ester linkage, although this phenol has still not been detected. The 5-position of the phenyl ring is selectively metabolized in insects and their microsomes.
[Aizawa, H. Metabolic Maps of Pesticides. New York, NY: Academic Press, 1982. 114]**PEER REVIEWED**

 

Absorption, Distribution & Excretion:

LIKE HOUSEFLIES, RAT ... DEGRADES ARPROCARB ... WITH 30% OF APPLIED DOSE EXPIRED AS CO2 WITHIN 48-HR ... .
[White-Stevens, R. (ed.). Pesticides in the Environment: Volume 1, Part 1, Part 2. New York: Marcel Dekker, Inc., 1971. 197]**PEER REVIEWED**

WHEN FED TO HUMANS, BAYGON WAS RECOVERED FROM URINE ALMOST QUANTITATIVELY AS O-ISOPROPOXYPHENOL, PROBABLY EXCRETED AS GLUCURONIDE ... .
[Menzie, C.M. Metabolism of Pesticides. U.S. Department of the Interior, Bureau of Sport Fisheries and Wildlife, Publication 127. Washington, DC: U.S. Government Printing Office, 1969. 50]**PEER REVIEWED**

Using published human data on skin to urine and blood to urine transfer of /propoxur/, ... the skin to blood transfer rates were estimated by 2 numerical deconvolution techniques. Regular constrained deconvolution produced an estimated upper limit on cumulative dermal absorption of the radiolabel, while minimized deconvolution produced an upper bound on cumulative dermal absorption of the parent cmpd. Dermal absorption rate was largest within 8 hr of dosing for /propoxur/. ...
[Fisher HL et al; JAT J Appl Toxicol 5 (3): 163-77 (1985)]**PEER REVIEWED**

Carbamates are readily absorbed through the lungs, gastrointestinal tract, and skin. Absorption by the respiratory tract depends on the vapor pressure of individual insecticides; carbaryl and aldicarb display low inhalation toxicity because of their high pressures. The lungs readily absorb propoxur (Baygon).
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1077]**PEER REVIEWED**

The hazard presented by application of insecticides to indoor surfaces where dermal exposure (and oral exposure in infants) may occur was investigated. Worst case assumptions for an infant playing in a room were used to calculate exposure for infant inhalation exposure, dermal exposure through contact with the floor and oral exposures due to hand/mouth contact. Surface exposure and total absorption were calculated for chlorpyrifos, dichlorvos, and propoxur. The dose calculated with each of these insecticides might reach toxic levels, particularly to an infant. Dose calculations did not consider metabolic breakdown or cumulative effects. Data necessary for determining that the levels of insecticides in the air and on treated surfaces will not be injurious to human health would include the bioavailability of surface residues, rate of transfer from surfaces and dose response data. According to the authors, risk assessments based on health protective assumptions should be used in the meantime to decide the safety of various components.
[Berteau PE et al; Biological Monitoring for Pesticide Exposure: Measurement, Estimation, & Risk Reduction, ACS Symp Series 382: 315-26 (1989)]**PEER REVIEWED**

 

Mechanism of Action:

Carbamylation of acetylcholinesterase produces accumulation of acetylcholine and the picture of muscarinic and nicotinic poisoning. Spontaneous hydrolysis of the carbamate-cholinesterase complex occurs in vivo, leading to the disappearance of clinical effects within 24 hours. Penetration of the blood-brain barrier by the carbamates is insignificant; for this reason, few CNS symptoms occur.
[Ellenhorn, M.J. and D.G. Barceloux. Medical Toxicology - Diagnosis and Treatment of Human Poisoning. New York, NY: Elsevier Science Publishing Co., Inc. 1988. 1077]**PEER REVIEWED**

 

Pharmacology:

 

 

Therapeutic Uses:

MEDICATION (VET): EFFECTIVE AGAINST FLEAS & TICKS ON CATTLE, HORSE, CATS, & DOGS ... & SARCOPTIC MANGE OF CATTLE ... PROTECTION AGAINST TICKS APPEARS TO WANE AFTER 1 WK.
[Rossoff, I.S. Handbook of Veterinary Drugs. New York: Springer Publishing Company, 1974. 493]**PEER REVIEWED**

 

Environmental Fate & Exposure:

 

 

Environmental Fate/Exposure Summary:

Propoxur, known chemically by o-isopropoxyphenyl N-methylcarbamate or 2-(1-methylethoxy)phenol methylcarbamate, is an insecticide and molluscide characterized by a fast knockdown and long residual effect. It will be released to the environment when it is applied as a spray or dust or used in bait to control household pests as well as lawn and garden insects. If released on soil it would not adsorb strongly to the soil. In one field study, 75% of propoxur disappeared from sandy soil in 100 days but levels were virtually unchanged in muck and silt loam soils. However, the rate of biodegradation is markedly increased when the soil has been previously exposed to methylcarbamate pesticides. Additionally, abiotic hydrolysis should be important in alkaline soils. Propoxur is readily degradable in water (half-life from 1 day to 1 week). In surface layers of water it will photolyze relatively rapidly, especially when humic material is present (half-life 13 - 88 hr). Degradability increases with temperature, high microbial populations, the presence of mud and biota, and increasing pH. Volatilization, adsorption to sediment and bioconcentration in fish should not be important fate processes. Propoxur would be released into the atmosphere primarily as a dust or aerosol and be subject to gravitational settling. The vapor phase chemical should react with photochemically produced hydroxyl radicals and have a half-life of about 4 hr. People will primarily be exposed to propoxur where it is used to control insects, both indoors and outdoors. (SRC)
**PEER REVIEWED**

 

Probable Routes of Human Exposure:

People may be exposed to propoxur in indoor air via inhalation or dermal contact where it is used to control cockroaches, flies and mosquitoes and outdoors when it is used to control lawn and turf insects(1). Exposure would be particularly high indoors(SRC).
[(1) Lewis RG et al; Environ Monit Assess 10: 59-73 (1988)]**PEER REVIEWED**

Propoxur was detected in 6 of 9 personal air samples of retired or semiretired persons in a pilot project of pesticide exposure(3). The concentrations measured ranged up to 0.60 ug/cu m, 0.10 ug/cu m, mean. Outside these houses, on the patio or porch, 4 of the nine samples contained propoxur ranging up to 0.0039 ug/cu m, 0.0034 ug/cu m, mean.
[(1) Lewis RG et al; Environ Monit Assess 10: 59-73 (1988)]**PEER REVIEWED**

 

Artificial Pollution Sources:

Propoxur is a nonsystemic insecticide and molluscide which was introduced in 1959 in Germany(2,4). It is characterized as having a fast knockdown and long residual effect(3). It may be released to the environment during its manufacture, transport, formulation, use, and disposal, as well as from spills. According to a National Pesticide Usage database, it is estimated that 51,200 lbs of propoxur are consumed annually for pesticidal use(1). It is available as an emulsifiable concentrate, wettable powder, dust or bit and is recommended for use against household pests such as cockroaches, mosquitoes, and flies, for control of lawn and turf insects, aphids, wooly aphids, bugs, and leafhoppers(3).
[(1) Gianessi LP; A National Pesticide Usage Data Base. Washington DC: Resources for the Future (1986) (2) Hawley GG; Condensed Chem Dictionary 10th ed Von Nostrand Reinhold NY (1981) (3) Meister RT; Farm Chemicals Handbook. p. C212 Willoughby, OH: Meister Publishing Co (1987) (4) Rajagopal BS et al; Res Rev 93: 1-199 (1984)]**PEER REVIEWED**

 

Environmental Fate:

/TERRESTRIAL FATE:/ 25% OF APPLIED BAYGON WAS LOST FROM SAND IN 100 DAYS, BUT ... PRACTICALLY NO DECLINE IN SILT-LOAM SOIL DURING 6 MO PERIOD
[Menzie, C.M. Metabolism of Pesticides. U.S. Department of the Interior, Bureau of Sport Fisheries and Wildlife, Publication 127. Washington, DC: U.S. Government Printing Office, 1969. 50]**PEER REVIEWED**

TERRESTRIAL FATE: Propoxur is not strongly adsorbed to soil and is highly persistent in neutral and acid soils. While 75% of the insecticide degraded in 100 days in a sandy soil, in a muck and silt loam soil, virtually no loss occurred during the same period and the propoxur persisted in these soils beyond six months(1). Other studies show that when the resident microorganisms are acclimated to methylcarbamate pesticides, biodegradation is markedly enhanced and in one study propoxur had largely disappeared in one day. Since propoxur hydrolyzes in basic media (half-life 16 days at pH 8), this process would be expected to contribute to propoxur loss in alkaline soil(SRC).
[(1) Rajagopal BS et al; Res Rev 93: 1-199 (1984)]**PEER REVIEWED**

AQUATIC FATE: Propoxur was found to be readily degradable in water. It was degraded in a model aquatic ecosystem containing the standard food chain members, algae, daphnia, mosquito larvae, snails, and fish(3), and highly biodegradable in components of a model terrestrial-aquatic ecosystem containing the same food chain members in the aqueous compartment(1). After 1 month, observed concentrations of propoxur were: 0.2 ppb in water, 15 ppb in snails, 50 ppb in fish, and traces in mosquito larvae(1). It was the second most labile of 9 pesticides dosed in lighted, agitated river water. The half-lives of the 9 pesticides ranged from 0.42 for carbaryl to 11.91 for lindane; the specific half-life of propoxur was not reported(2). The degradation rates were temperature dependent and altered by the presence of mud, aquatic plants and invertebrates(2). Propoxur has also been shown to photolyze in waters (half-life 88 hr), and this photolysis is enhanced by the presence of humic material (half-life 13 - 41 hr)(4). Therefore photolysis may be a major factor affecting propoxur disappearance in surface water as exemplified by a 1 week half-life in water from the Little Miami River(5).
[(1) Sangha GK; Diss Abstr Int B 32: 4650 (1971) (2) Bogacka T; Rocz Panstw Zakl Hig 33: 281-9 (1982) (3) Lu PY; Diss Abstr Int B 35: 303 (1974) (4) Jensen-Korte U et al; Sci Tot Environ 62: 335-40 (1987) (5) Eichelberger JW, Lichtenberg JJ; Environ Sci Technol 5: 541-4 (1971)]**PEER REVIEWED**

ATMOSPHERIC FATE: Propoxur would be primarily released to the atmosphere in the form of a dust or aerosol during its use as an insecticide and would be subject to gravitational settling. However propoxur also has been shown to volatilize from baits. Vapor phase propoxur resulting from this volatilization is estimated to have a half-life of 4.3 hr with an average ambient hydroxyl radical concentration of 5X10+5 radicals/cc(1). However indoors, the half-life should be much longer because the concentration of hydroxyl radicals is lower(SRC).
[(1) Atkinson R; Int J Chem Kinet 19: 799-828 (1987)]**PEER REVIEWED**

 

Environmental Biodegradation:

Degradation rates of carbofuran were greatly increased by a single 10 ppm carbofuran pretreatment of a sandy loam soil with no previous history of pesticide use. Thorough mixing was required to homogeneously distribute the active agent(s). Numbers of bacteria and fungi were not affected by the pretreatment, but the marked decrease in activity induced by heat sterilization, freezing, or drying suggests that soil microorganisms are the active agents. Increased degradation rates for a variety of aryl- and oximino-methyl carbamates were observed /including propoxur/.
[Harris CR et al; J Environ Sci Health, Part B; B19(1): 1-11 (1984)]**PEER REVIEWED**

Propoxur is reported to biodegrades quite rapidly in water, particularly when the bacterial activity and temperature is high(1). In a biodegradation test which used a combination of activated sludge, silt loam soil, and sediment as an inoculum, the half-life of propoxur was 44 days under aerobic conditions and 59 days under anaerobic conditions(3). When glucose and peptone was added as a source of carbon and energy, the half-life decreased to 19 days under aerobic conditions and increased slightly under anaerobic conditions. When 10 ppb propoxur was added to water from the Little Miami River (Ohio), a river receiving both domestic and industrial waste and incubated in closed jars under sunlight and artificial fluorescent light, 50, 70, 90, and 95% was hydrolyzed to its phenol after 1, 2, 4, and 8 weeks, respectively(2). Small amounts of propoxur were still present in the water after 8 weeks. The pH of Miami River water was 7.3 at the start of the experiment but varied up to 8.0 during the eight-week period. Therefore chemical hydrolysis may have contributed to the degradation(SRC).
[(1) Gummer WD; in Water Quality Interpretive Report No. 4. Regina, Saskatchewan, Canada: Inland Waters Directorate (1979) (2) Eichelberger JW, Lichtenberg JJ; Environ Sci Technol 5: 541-4 (1971) (3) Kanazawa J; Environ Monit Asses 9: 57-70 (1987)]**PEER REVIEWED**

One day after propoxur was applied to a sandy loam soil with no history of insecticidal treatment, 94% remained(1). However when the soil was previously treated four times with carbofuran, also a methylcarbamate pesticide, only 20% of the propoxur remained after 1 day. This shows that acclimation of resident microorganisms markedly affects the rate of biodegradation. 2-Isopropoxyphenol is a product of propoxur biodegradation(2).
[(1) Harris CR et al; J Environ Sci Health B19: 1-11 (1984) (2) Rajagopal BS et al; Res Rev 93: 1-199 (1984)]**PEER REVIEWED**

 

Environmental Abiotic Degradation:

HYDROLYZES @ RATE OF 1.5%/DAY IN 1% AQ @ PH 7.
[Spencer, E. Y. Guide to the Chemicals Used in Crop Protection. 7th ed. Publication 1093. Research Institute, Agriculture Canada, Ottawa, Canada: Information Canada, 1982. 488]**PEER REVIEWED**

The half-life of propoxur in water was determined to be 87.9 hr when irradiated with light >290nm(1). When 10 and 100 ppm of humic acids were added the half-life decreased to 40.8 and 13.0 hr, respectively(1). It was observed when irradiating solutions of propoxur with a germicidal lamp (peak wavelength 254 nm), that photodecomposition proceeded at increasing rates as the pH was increased from 5 to 9; the half-life decreased from 19.0 to 9.0 minutes(5). The primary effect of irradiation is the cleavage of the ester bond and formation of the phenol(5). The hydrolysis of propoxur was studied at 20 deg C over a range of pHs(4). It was stable between pH 3.0 and 7.0, however at pH 8, 9, and 10 hydrolysis was observed and the respective half-lives were 16, 1.6, and 0.17 days(4). Furthermore, the rate of hydrolysis increases by a factor of 2.49 for each 10 deg rise in temperature(4). Propoxur is unstable in alkaline media, having a half-life of 40 minutes at pH 10 and 20 deg C(2). It was relatively stable over the course of 14 days at pH 6.9 when used as a control in a biodegradation experiment, having a half-life of 78 days under aerobic conditions and 124 days under anaerobic conditions(3). 2-Isopropoxyphenol is formed during hydrolysis(2).
[(1) Jensen-Korte U et al; Sci Tot Environ 62: 335-40 (1987) (2) Pesticide Manual; 5th ed. p. 445 British Crop Protection Council (1977) (3) Kanazawa J; Environ Monit Asses 9: 57-70 (1987) (4) Aly OM, El-Dib MA; Water Res 5: 1191- 205 (1971) (5) Aly OM, El-Dib MA; in Fate of Organic Pesticides in the Aquatic Environment. Adv Chem Series III. Chapt 11 pp.210-33 (1972)]**PEER REVIEWED**

Vapor phase propoxur will degrade in the atmosphere by H-atom extraction and addition to the aromatic ring by photochemically produced hydroxyl radicals. The estimated half-life resulting from these reactions is 4.3 hr with a hydroxyl radical concentration of 5X10+5 radicals/cc(1). However indoors, the half-life should be much longer because the concentration of hydroxyl radicals is lower(SRC).
[(1) Atkinson R; Int J Chem Kinet 19: 799-828 (1987)]**PEER REVIEWED**

 

Environmental Bioconcentration:

Bioconcentration factor = 9 (calculated from water solubility by regression equations). /From table/
[Kenaga EE; Ecotoxicology and Environmental Safety 4: 31 (1980)]**PEER REVIEWED**

Using the recommended water octanol/water partition coefficient for propoxur, 1.52(1), one estimates a BCF of 8.4 using a recommended regression equation(2). Therefore propoxur would not be expected to bioconcentrate significantly in fish and aquatic organisms.
[(1) Hansch C, Leo AJ; MEDCHEM Project Claremont CA: Pomona College (1985) (2) Lyman WJ et al; pp. 5-1 to 5-34 Handbook of Chemical Property Estimation Methods New York: McGraw-Hill (1982)]**PEER REVIEWED**

 

Soil Adsorption/Mobility:

KOC = 67 (calculated from water solubility by regression equations). /From table/
[Kenaga EE; Ecotoxicology and Environmental Safety 4: 31 (1980)]**PEER REVIEWED**

Using the water solubility, for propoxur, 1750 ppm(2), one estimates a Koc of 72 using a recommended regression equation(3). Therefore propoxur would not be expected to adsorb significantly to organic soil. The adsorption of propoxur to various clays was found to conform with the Freundlich adsorption equation(1). The Freundlich adsorption constant, K was 0.038 to kaolinite and 0.060 to bentonite and the 1/N exponents in the Freundlich equation were 0.37 and 0.52. The adsorption isotherm is therefore markedly nonlinear and the adsorptivity low(1).
[(1) Aly OM, El-Dib MA; in Fate of Organic Pesticides in the Aquatic Environment. Adv Chem Series III. Chapt 11 pp. 210-33 (1972) (2) Meister RT; Farm Chemicals Handbook. p. C212 Willoughby, OH: Meister Publishing Co (1987) (3) Lyman WJ et al; pp. 4-1 to 4-34 Handbook of Chemical Property Estimation Methods New York: McGraw-Hill (1982)]**PEER REVIEWED**

Soil sorption constants based on the organic carbon content of 15 pesticides were measured using 2 soils (clay loam and high clay) at 0.01, 0.1 and 1.0 ppm pesticide. The soil sorption coefficients ((ug pesticide/g soil)/(ug pesticide/g water)) for propoxur were 1.7 + or - 0.8 in clay loam and 0.8 + or - 0.3 in high clay soil. The soil sorption constants were 41 and 53 respectively, with a mean of 47. Significant correlations were found between organic carbon content and water solubility, octanol/water partition coefficient, retention time in reversed phase high pressure liquid chromatography and molecular wt.
[Kanazawa J; Environ Toxicol Chem 8 (6): 477-84 (1989)]**PEER REVIEWED**

 

Volatilization from Water/Soil:

The Henry's law constant for propoxur calculated from its vapor pressure, 3X10-6 mm Hg at 20 deg C(1), and water solubility, 1750 ppm(2), is 0.444X10-9 atm-cu m/mol. Volatilization from water for compounds with such low Henry's law constants would be negligible.
[(1) Wright CG et al; Bull Environ Contam Toxicol 26: 548-53 (1981) (2) Meister RT; Farm Chemicals Handbook. p. C212 Willoughby, OH: Meister Publishing Co (1987)]**PEER REVIEWED**

 

Environmental Water Concentrations:

SURFACE WATER: After propoxur was aerially applied around Winnipeg, Manitoba for mosquito control, half of the samples collected from surface water in and around the city was positive for the insecticide(1). The highest level was 36.4 ppb, in a creek 12 hr after spraying. Four other samples were in excess of 17.5 ppb.
[(1) Gummer WD; in Water Quality Interpretive report No. 4. Regina, Saskatchewan, Canada: Inland Waters Directorate (1979)]**PEER REVIEWED**

 

Atmospheric Concentrations:

INDOOR AIR: The airborne concentration of propoxur after application as a 1.1% emulsion to a 61.2 cu m dormitory room whose approximate temperature and humidity was 25 deg C and 60%, respectively was 15.4 ug/cu m(1). After 1, 2, and 3 days, the levels declined to 2.7, 1.8, and 0.7 ug/cu m, respectively. Prior to treatment, no propoxur was detectable. In another study, 36 2.5 x 1.0 cm insecticide strips containing 10% propoxur were placed in a 30 cu m room maintained at 25 deg C and 50% relative humidity and ventilated at 8-10 air exchanges per hour(2). Half of the strips were placed on the floor and half attached to the wall at 1 m height. The strips produced a vapor concentration which ranged between 0.33 and 0.79 ug/cu m over the 30 day period in which sampling was conducted(2). Propoxur was detected in the indoor air of 7 of 9 households in a pilot project of pesticide exposure(3). Areas of high household activity such as the kitchen were sampled. The concentrations measured ranged from not detected to 0.31 ug/cu m, 0.042 ug/cu m, mean. Outside these houses, on the patio or porch, 4 of the nine samples contained propoxur ranging up to 0.0039 ug/cu m, 0.0034 ug/cu m, mean.
[(1) Wright CG et al; Bull Environ Contam Toxicol 26: 548-53 (1981) (2) Jackson MD, Lewis RG; Bull Environ Contam Toxicol 27: 122-5 (1981) (3) Lewis RG et al; Environ Monit Assess 10: 59-73 (1988)]**PEER REVIEWED**

 

Food Survey Values:

A non-random sampling of 39 different fresh crops, dehydrated apples, crackers, and wines analyzed for carbamate residues (319 samples), found 1 sample of rice and 1 of crackers containing propoxur residue of 8.1 and 0.08 ppm, respectively(1).
[(1) Krause RT; J Assoc Off Analyt Chem 68: 734-41 (1985)]**PEER REVIEWED**

 

Environmental Standards & Regulations:

 

 

Acceptable Daily Intakes:

FAO/WHO ADI: 0.02 mg/kg bw
[FAO/WHO; Pesticide Residues in Food - 1991. Joint Meeting of the FAO Panel of Experts on Pesticide Residues in Food and the Environment and the WHO Expert Group on Pesticide Residues. Geneva, September 16-22, 1991. Evaluations Part 1 - Residues. p.640 FAO Plant & Prod Protect Paper 113/1 (1992)]**QC REVIEWED**

 

Atmospheric Standards:

Listed as a hazardous air pollutant (HAP) generally known or suspected to cause serious health problems. The Clean Air Act, as amended in 1990, directs EPA to set standards requiring major sources to sharply reduce routine emissions of toxic pollutants. EPA is required to establish and phase in specific performance based standards for all air emission sources that emit one or more of the listed pollutants. Propoxur is included on this list.
[Clean Air Act as amended in 1990, Sect. 112 (b) (1) Public Law 101-549 Nov. 15, 1990]**QC REVIEWED**

 

Federal Drinking Water Guidelines:

EPA 3 ug/l
[USEPA/Office of Water; Federal-State Toxicology and Risk Analysis Committee (FSTRAC). Summary of State and Federal Drinking Water Standards and Guidelines (11/93)] **QC REVIEWED**

 

State Drinking Water Guidelines:

(AZ) ARIZONA 3 ug/l
[USEPA/Office of Water; Federal-State Toxicology and Risk Analysis Committee (FSTRAC). Summary of State and Federal Drinking Water Standards and Guidelines (11/93)] **QC REVIEWED**

(CA) CALIFORNIA 90 ug/l
[USEPA/Office of Water; Federal-State Toxicology and Risk Analysis Committee (FSTRAC). Summary of State and Federal Drinking Water Standards and Guidelines (11/93)] **QC REVIEWED**

(FL) FLORIDA 28 ug/l
[USEPA/Office of Water; Federal-State Toxicology and Risk Analysis Committee (FSTRAC). Summary of State and Federal Drinking Water Standards and Guidelines (11/93)] **QC REVIEWED**

(ME) MAINE 3 ug/l
[USEPA/Office of Water; Federal-State Toxicology and Risk Analysis Committee (FSTRAC). Summary of State and Federal Drinking Water Standards and Guidelines (11/93)] **QC REVIEWED**

 

Chemical/Physical Properties:

 

 

Molecular Formula:

C11-H15-N-O3
**PEER REVIEWED**

 

Molecular Weight:

209.24
[Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989. 1245]**PEER REVIEWED**

 

Color/Form:

MINUTE CRYSTALS
[Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989. 1245]**PEER REVIEWED**

WHITE TO TAN CRYSTALLINE SOLID
[Farm Chemicals Handbook 1991. Willoughby, OH: Meister, 1991.,p. C-253]**PEER REVIEWED**

White to tan, crystalline powder.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 94-116. Washington, D.C.: U.S. Government Printing Office, June 1994. 266]**QC REVIEWED**

 

Odor:

Faint, characteristic odor.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 94-116. Washington, D.C.: U.S. Government Printing Office, June 1994. 266]**QC REVIEWED**

 

Melting Point:

91.5 DEG C
[Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989. 1245]**PEER REVIEWED**

 

Octanol/Water Partition Coefficient:

log Kow = 1.52
[Hansch, C., Leo, A., D. Hoekman. Exploring QSAR - Hydrophobic, Electronic, and Steric Constants. Washington, DC: American Chemical Society., 1995. 88]**QC REVIEWED**

 

Solubilities:

SOL IN METHANOL, ACETONE, & MANY ORG SOLVENTS; SLIGHTLY SOL IN COLD HYDROCARBONS; SOL IN WATER ABOUT 0.2% @ 20 DEG C
[Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989. 1245]**PEER REVIEWED**

water solubility = 1860 mg/l @ 30 deg C
[Bowman BT, Sans WW; Further Water Solubility Determination of Insecticidal Compounds. J Environ Sci Health B18:221-7 (1983)]**QC REVIEWED**

 

Spectral Properties:

Intense mass spectral peaks: 110 m/z (100%), 152 m/z (18%), 111 m/z (10%), 81 m/z (10%)
[Hites, R.A. Handbook of Mass Spectra of Environmental Contaminants. Boca Raton, FL: CRC Press Inc., 1985. 225]**PEER REVIEWED**

Intense mass spectral peaks: 209 m/z
[Pfleger, K., H. Maurer and A. Weber. Mass Spectral and GC Data of Drugs, Poisons and their Metabolites. Parts I and II. Mass Spectra Indexes. Weinheim, FederalRepublic of Germany. 1985. 320]**PEER REVIEWED**

 

Vapor Pressure:

3X10-6 mm Hg at 20 deg C
[Wright CG et al; Bull Environ Contam Toxicol 26: 548-53 (1981)]**PEER REVIEWED**

 

Other Chemical/Physical Properties:

DECOMP @ HIGH TEMP FORMING METHYL ISOCYANATE
[Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989. 1245]**PEER REVIEWED**

Vapor pressure: 1.3 Pa @ 120 Deg C
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 717]**PEER REVIEWED**

 

Chemical Safety & Handling:

 

 

Hazardous Reactivities & Incompatibilities:

Believed compatible with most insecticides, fungicides, except alkalines.
[Farm Chemicals Handbook 1991. Willoughby, OH: Meister, 1991.,p. C-253]**PEER REVIEWED**

Strong oxidizers, alkalis [Note: Emits highly toxic methyl isocyanate fumes when heated to decomposition].
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

 

Hazardous Decomposition:

DANGEROUS. WHEN HEATED TO DECOMP IT EMITS HIGHLY TOXIC FUMES. /SRP: OF METHYL ISOCYANATE/
[Sax, N.I. Dangerous Properties of Industrial Materials. 4th ed. New York: Van Nostrand Reinhold, 1975. 438]**PEER REVIEWED**

 

Protective Equipment & Clothing:

PROTECTIVE MEASURES SHOULD INCL CLEAN OVERALLS DAILY, ... IMPERVIOUS SHOES, RUBBER GLOVES (MIXER ONLY), WASHING OF HANDS & FACE FOLLOWING EACH PUMP CHARGE ... .
[Hayes, W. J., Jr. Toxicology of Pesticides Baltimore: Williams & Wilkins, 1975. 332]**PEER REVIEWED**

Wear appropriate personal protective clothing to prevent skin contact.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

Wear appropriate eye protection to prevent eye contact.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

 

Preventive Measures:

SRP: Contaminated protective clothing should be segregated in such a manner so that there is no direct personal contact by personnel who handle, dispose, or clean the clothing. Quality assurance to ascertain the completeness of the cleaning procedures should be implemented before the decontaminated protective clothing is returned for reuse by the workers. Contaminated clothing should not be taken home at end of shift, but should remain at employee's place of work for cleaning.
**PEER REVIEWED**

The worker should wash daily at the end of each work shift.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

Work clothing that becomes wet or significantly contaminated should be removed and replaced.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

Workers whose clothing may have become contaminated should change into uncontaminated clothing before leaving the work premises.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

 

Stability/Shelf Life:

UNSTABLE IN HIGHLY ALKALINE MEDIA, 50% LOSS @ 20 DEG C IN 40 MIN @ pH 10
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 717]**PEER REVIEWED**

 

Shipment Methods and Regulations:

No person may /transport,/ offer or accept a hazardous material for transportation in commerce unless that person is registered in conformance ... and the hazardous material is properly classed, described, packaged, marked, labeled, and in condition for shipment as required or authorized by ... /the hazardous materials regulations (49 CFR 171-177)./
[49 CFR 171.2 (7/1/96)]**QC REVIEWED**

The International Maritime Dangerous Goods Code lays down basic principles for transporting hazardous chemicals. Detailed recommendations for individual substances and a number of recommendations for good practice are included in the classes dealing with such substances. A general index of technical names has also been compiled. This index should always be consulted when attempting to locate the appropriate procedures to be used when shipping any substance or article.
[IMDG; International Maritime Dangerous Goods Code; International Maritime Organization p.3097-1,6193,6194,6195 (1988)]**QC REVIEWED**

 

Storage Conditions:

Store in original container, preferably in locked area, away from children, food, feed.
[Farm Chemicals Handbook 1991. Willoughby, OH: Meister, 1991.,p. C-253]**PEER REVIEWED**

 

Cleanup Methods:

Facultative and aerobic digesters for livestock wastes were used to degrade pesticide materials, ie ... Baygon, in the lab. Three different animal wastes were used as digester media for the process: swine, chicken, and dairy cattle manure. Process effects on disappearance of /Baygon/ was observed over periods of 0-8 wk (facultative) and 0-6 wk (aerobic). Disappearance ranged 10-72% under facultative conditions, and 29-100% under aerobic conditions. ...
[Collins ER Jr et al; Trans ASAE 27 (3): 755-61 (1984)]**PEER REVIEWED**

The Carbolator 35B, a recirculatory C filtration system, successfully treated pesticide wastes in both pilot-scale and field tests. In pilot-scale tests, the system was challenged with 400 gal of water containing 20, 60, and 100 mg/l of Baygon. The pesticide waste was pumped through the Carbolator and returned to the waste holding tank. The tank contents were analyzed by gas chromatography and TLC. A TLC method was developed to perform pesticide anal in the field. A simple lab recirculating system using 4 liter of waste was also built to simulate the Carbolator system. A math model was developed to predict the disappearance of the pesticides from the waste holding tank.
[Kobylinski EA et al; ACS Symp Ser 259: 125-51 (1984)]**PEER REVIEWED**

 

Disposal Methods:

Hydrolysis & landfill: Propoxur is hydrolyzed in highly alkaline media, with 50% loss at 20 deg C in 40 min at pH 10. It should be treated with alkali before burial. Recommendable method: Incineration. Peer-review: Large amt should be incinerated in a unit with effluent gas scrubbing. (Peer-review conclusions of an IRPTC expert consultation (May 1985))
[United Nations. Treatment and Disposal Methods for Waste Chemicals (IRPTC File). Data Profile Series No. 5. Geneva, Switzerland: United Nations Environmental Programme, Dec. 1985. 128]**PEER REVIEWED**

 

Occupational Exposure Standards:

 

 

OSHA Standards:

Vacated 1989 OSHA PEL TWA 0.5 mg/cu m is still enforced in some states.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 370]**QC REVIEWED**

 

Threshold Limit Values:

8 hr Time Weighted Avg (TWA): 0.5 mg/cu m
[American Conference of Governmental Industrial Hygienists. Threshold Limit Values (TLVs) for Chemical Substances and Physical Agents Biological Exposure Indices for 1998. Cincinnati, OH: ACGIH, 1998. 58]**QC REVIEWED**

Excursion Limit Recommendation: Excursions in worker exposure levels may exceed three times the TLV-TWA for no more than a total of 30 min during a work day, and under no circumstances should they exceed five times the TLV-TWA, provided that the TLV-TWA is not exceeded.
[American Conference of Governmental Industrial Hygienists. Threshold Limit Values (TLVs) for Chemical Substances and Physical Agents Biological Exposure Indices for 1998. Cincinnati, OH: ACGIH, 1998. 6]**QC REVIEWED**

A3. A3= Confirmed animal carcinogen with unknown relevance to humans.
[American Conference of Governmental Industrial Hygienists. Threshold Limit Values (TLVs) for Chemical Substances and Physical Agents Biological Exposure Indices for 1998. Cincinnati, OH: ACGIH, 1998. 58]**QC REVIEWED**

 

NIOSH Recommendations:

Recommended Exposure Limit: 10 Hr Time-Weighted Avg: 0.5 mg/cu m.
[NIOSH. NIOSH Pocket Guide to Chemical Hazards. DHHS (NIOSH) Publication No. 97-140. Washington, D.C. U.S. Government Printing Office, 1997. 266]**QC REVIEWED**

 

Manufacturing/Use Information:

 

 

Major Uses:

Insecticide; Molluscicide
[Sax, N.I. and R.J. Lewis, Sr. (eds.). Hawley's Condensed Chemical Dictionary. 11th ed. New York: Van Nostrand Reinhold Co., 1987. 660]**PEER REVIEWED**

BAYGON*: EFFECTIVE AGAINST COCKROACHES, FLIES, MOSQUITOES ... . ALSO ... FOR CONTROL OF WOOLY APHIDS, BUGS, & LEAF HOPPERS.
[Farm Chemicals Handbook 1991. Willoughby, OH: Meister, 1991.,p. C-253]**PEER REVIEWED**

MEDICATION (VET)
**PEER REVIEWED**

 

Manufacturers:

MOBAY CHEM CORP, CHEMAGRO AGRICULTURAL DIV, KANSAS CITY, MO
[SRI]**PEER REVIEWED**

 

Methods of Manufacturing:

REACTION OF O-ISOPROPOXYPHENOL AND METHYL ISOCYANATE
[SRI]**PEER REVIEWED**

 

General Manufacturing Information:

NON-PHYTOTOXIC
[Worthing, C. R. (ed.). Pesticide Manual. 6th ed. Worcestershire, England: British Crop Protection Council, l979. 452]**PEER REVIEWED**

Its use for malaria control has tended to emphasize /propoxur's/ vapor toxicity to mosquitoes. Test mosquitoes may be killed by overnight exposure in a cage in the center of a room as long as 30 weeks after spraying. In villages with houses close together, the lethal effect soon after spraying may extend outdoors from house to house in the entire center of the village and decline only gradually on the edges of the village. This vapor action appears advantageous in areas where malaria mosquitoes enter houses and bite people but do not rest long enough on sprayed surfaces to acquire a lethal dose or in villages where considerable outdoor biting occurs.
[Hayes, Wayland J., Jr. Pesticides Studied in Man. Baltimore/London: Williams and Wilkins, 1982. 448]**PEER REVIEWED**

Formulations/Preparations:

Emulsifiable concentrate, wettable powder, dustable powder, granular, aerosol generator, smoke generator and bait /formulations/ of different ai concentrations.
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 717]**PEER REVIEWED**

Consumption Patterns:

OVER 50% AS AN INSECTICIDE FOR MOSQUITOES IN OUTDOOR AREAS; UNDER 50% IN ALL OTHER APPLICATIONS AS AN INSECTICIDE (FOR FLIES ON AGRICULTURAL PREMISES, FOR FLEAS & TICKS ON PETS, ON LAWNS & TURF, FOR ANTS ON FLOWERING PLANTS, & IN DWELLINGS & PUBLIC FACILITIES) (1974)
[SRI]**PEER REVIEWED**

U. S. Production:

(1972) PROBABLY GREATER THAN 4.54X10+5 GRAMS
[SRI]**PEER REVIEWED**

(1974) 1.4X10+8 GRAMS (CONSUMPTION)
[SRI]**PEER REVIEWED**

U. S. Imports:

(1974) 1.4X10+8 GRAMS (EST)
[SRI]**PEER REVIEWED**

Laboratory Methods:

Analytic Laboratory Methods:

... UV SPECTROSCOPY AFTER HYDROLYSIS TO 2-ISOPROPOXYPHENOL OR BY IR SPECTROSCOPY. RESIDUES MAY BE DETERMINED BY GLC OR, AFTER HYDROLYSIS, BY COLORIMETRIC ESTIMATION OF 2-ISOPROPOXYPHENOL.
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 718]**PEER REVIEWED**

A procedure is described for the assay of propoxur by NMR spectroscopy. Methanol was used as known std and tech materials were analyzed and the results of NMR and liquid chromatography methods were compared. The NMR method is straightforward, with a precision of + or - 1.6% (95% confidence limits) for 2 sample weighings.
[Lesser JH et al; J Assoc Off Anal Chem 68 (3): 565-7 (1985)]**PEER REVIEWED**

Four laboratories obtained 177 carbamate recovery values using a liquid chromatographic method. The average recovery of 11 carbamates ... from 14 crops was 99% with a coefficient of variation of 8% (0.03-1.8 ppm fortification levels). ... A fifth laboratory contributed 34 carbamate recoveries (average 99%) on table-ready food products for 4 carbamates. Propoxur is quantitatively recovered through the method. ...
[Krause RT; J Assoc Off Anal Chem 68 (4): 734-41 (1985)]**PEER REVIEWED**

A liquid chromatographic method for the determination of propoxur in technical and formulated products has been subjected to a collaborative study with 8 participating collaborators. Formulations were extended with methyl cyanate and analyzed by reversed-phase chromatography with n-butyrophenone as an internal standard. ... Coefficient of variation values obtained on the 4 samples were 0.34, 0.68, 3.25, and 5.41% repectively.
[Slahck SC; J Assoc Off Anal Chem 67 (3): 497-9 (1984)]**PEER REVIEWED**

Product analysis is by hydrolysis and titration of the liberated methylamine; by ultra violet spectrometry after hydrolysis to 2-isopropoxyphenol or by infared spectrometry.
[Worthing, C.R., S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 7th ed. Lavenham, Suffolk, Great Britain: The Lavenham Press Limited, 1983. 470]**PEER REVIEWED**

The analyte can be extracted and can be chromatographed as determined by Athen-ERL or S-cubed.
[USEPA/OST; List of Lists: A Catalog of Analytes and Methods p.444 (1991) OST Pub 21W-4005]**PEER REVIEWED**

Sampling Procedures:

The insecticide propoxur can be collected at lower levels of 0.1 ppb using a common gas chromatographic packing material. The method is simplified by the use of sorbent tubes containing 10% Carbowax 400 on 80/100 mesh Supelcoport. Percent recovery was determined by drawing air across a polyvinyl chloride filter spiked with 0.5 or 1.0 mg of propoxur connected to a sorbent tube. No detectable levels were observed in a ventilated area. To evaluate the effect of storage, some samples were refrigerated 2 to 3 weeks, and others were collected in an unventilated storage room treated with propoxur. No significant difference was found between refrigerated samples and storage room samples when they were analyzed.
[Vasta JF; Kvartek EJ; American Industrial Hygiene Association Journal 46 (4): 230-232 (1985)]**PEER REVIEWED**

Sampling efficiency of five solid sorbents for trapping airborne pesticides,
[Roper EM, Wright CG; Bull Environ Contam Toxicol 33 (4): 476-83 (1984)]**PEER REVIEWED**

Special References:

Synonyms and Identifiers:

Related HSDB Records:

602 [ISOPROPYL PHENYLCARBAMATE]

Synonyms:

58 12 315
**PEER REVIEWED**

Aprocarb
**PEER REVIEWED**

ARPROCARB
**PEER REVIEWED**

Bay 39007
**PEER REVIEWED**

BAY 5122
**PEER REVIEWED**

Bay 9010
**PEER REVIEWED**

BAYER B 5122
**PEER REVIEWED**

BAYER 39007
**PEER REVIEWED**

Baygon
**PEER REVIEWED**

Bifex
**PEER REVIEWED**

Blattanex
**PEER REVIEWED**

Blattosep
**PEER REVIEWED**

Bolfo
**PEER REVIEWED**

Boruho
**PEER REVIEWED**

Brygou
**PEER REVIEWED**

CARBAMIC ACID, METHYL-, O-ISOPROPOXYPHENYL ESTER
**PEER REVIEWED**

Carbamic acid, methyl-, 2-(1-methylethoxy)phenyl ester
**PEER REVIEWED**

Chemagro 9010
**PEER REVIEWED**

DALF DUST
**PEER REVIEWED**

ENT 25,671
**PEER REVIEWED**

ENT-25671
**PEER REVIEWED**

o-IMPC
**PEER REVIEWED**

INVISI-GARD
**PEER REVIEWED**

IPMC
**PEER REVIEWED**

ISOCARB
**PEER REVIEWED**

2-ISOPROPOXYPHENYL N-METHYLCARBAMATE
**PEER REVIEWED**

O-ISOPROPOXYPHENYL METHYLCARBAMATE
**PEER REVIEWED**

O-ISOPROPOXYPHENYL N-METHYLCARBAMATE
**PEER REVIEWED**

O-(2-ISOPROPOXYPHENYL) N-METHYLCARBAMATE
**PEER REVIEWED**

2-ISOPROPOXYPHENYL METHYLCARBAMATE
**PEER REVIEWED**

2-(1-Methylethoxy)phenol methylcarbamate
**PEER REVIEWED**

MROWKOZOL
**PEER REVIEWED**

OMS 33
**PEER REVIEWED**

PHC
**PEER REVIEWED**

PHENOL, 2-(1-METHYLETHOXY)-, METHYLCARBAMATE
**PEER REVIEWED**

Propoksuru (Polish)
**PEER REVIEWED**

PROPOTOX
**PEER REVIEWED**

Propoxure
**PEER REVIEWED**

Propyon
**PEER REVIEWED**

Rhoden
**PEER REVIEWED**

Sendran
**PEER REVIEWED**

Suncide
**PEER REVIEWED**

Tendex
**PEER REVIEWED**

Tugon Fliegenkugel
**PEER REVIEWED**

Unden
**PEER REVIEWED**

Formulations/Preparations:

Emulsifiable concentrate, wettable powder, dustable powder, granular, aerosol generator, smoke generator and bait /formulations/ of different ai concentrations.
[Worthing, C.R. and S.B. Walker (eds.). The Pesticide Manual - A World Compendium. 8th ed. Thornton Heath, UK: The British Crop Protection Council, 1987. 717]**PEER REVIEWED**

RTECS Number:

NIOSH/FC3150000

Administrative Information:

Hazardous Substances Databank Number: 603
Last Revision Date: 20010808
Last Review Date: Reviewed by SRP on 11/07/1991
Update History:

Field Update on 08/08/2001, 1 field added/edited/deleted.
Field Update on 05/15/2001, 1 field added/edited/deleted.
Complete Update on 06/12/2000, 1 field added/edited/deleted.
Complete Update on 03/28/2000, 1 field added/edited/deleted.
Complete Update on 02/02/2000, 1 field added/edited/deleted.
Complete Update on 09/21/1999, 1 field added/edited/deleted.
Complete Update on 08/26/1999, 1 field added/edited/deleted.
Complete Update on 07/27/1999, 4 fields added/edited/deleted.
Complete Update on 03/29/1999, 2 fields added/edited/deleted.
Complete Update on 01/27/1999, 1 field added/edited/deleted.
Complete Update on 11/12/1998, 2 fields added/edited/deleted.
Complete Update on 06/02/1998, 1 field added/edited/deleted.
Complete Update on 03/16/1998, 7 fields added/edited/deleted.
Field Update on 10/17/1997, 1 field added/edited/deleted.
Field Update on 09/17/1997, 1 field added/edited/deleted.
Field Update on 08/12/1997, 5 fields added/edited/deleted.
Field Update on 05/08/1997, 1 field added/edited/deleted.
Field Update on 05/01/1997, 2 fields added/edited/deleted.
Complete Update on 10/12/1996, 1 field added/edited/deleted.
Complete Update on 09/04/1996, 6 fields added/edited/deleted.
Complete Update on 06/11/1996, 1 field added/edited/deleted.
Complete Update on 05/10/1996, 1 field added/edited/deleted.
Complete Update on 01/19/1996, 1 field added/edited/deleted.
Complete Update on 04/20/1995, 1 field added/edited/deleted.
Complete Update on 04/20/1995, 1 field added/edited/deleted.
Complete Update on 01/24/1995, 1 field added/edited/deleted.
Complete Update on 12/21/1994, 1 field added/edited/deleted.
Complete Update on 08/02/1994, 1 field added/edited/deleted.
Complete Update on 06/16/1994, 1 field added/edited/deleted.
Complete Update on 03/25/1994, 1 field added/edited/deleted.
Complete Update on 08/07/1993, 1 field added/edited/deleted.
Field update on 12/14/1992, 1 field added/edited/deleted.
Complete Update on 11/27/1992, 1 field added/edited/deleted.
Complete Update on 09/14/1992, 46 fields added/edited/deleted.
Complete Update on 08/17/1992, 46 fields added/edited/deleted.
Field Update on 01/13/1992, 1 field added/edited/deleted.
Complete Update on 11/01/1990, 18 fields added/edited/deleted.
Field Update on 05/14/1990, 1 field added/edited/deleted.
Field Update on 03/06/1990, 1 field added/edited/deleted.
Field Update on 01/15/1990, 1 field added/edited/deleted.
Complete Update on 01/11/1990, 17 fields added/edited/deleted.
Field Update on 02/10/1989, 1 field added/edited/deleted.
Complete Update on 03/08/1988, 2 fields added/edited/deleted.
Complete Update on 11/21/1986
Record Length: 89800